To understand the reasons behind the higher occurrence of coronary heart disease in diabetes, a thorough investigation of how oxidized LDL from various population subgroups affects endothelium-dependent relaxation is necessary.: To understand the reasons behind the higher occurrence of coronary heart disease in diabetes, a thorough investigation of how oxidized LDL from various population subgroups affects endothelium-dependent relaxation is necessary.

XIONGZIZENG XIONGZIZENG; DR SURIYAKALA SURIYAKALA

Authors

XIONGZIZENG PhD. Research Scholar in Medical Science, Lincoln University College, Malaysia
DR SURIYAKALA SURIYAKALA Professors in Lincoln University College, Malaysia

Keywords:

Cholesterol, Control Study, Effective Dose

Abstract

This study will not include Apolipoprotein B, and the possibility of a second coronary ischemia event will not be considered because it is a case-control study. Additionally, the atorvastatin dosage will not be increased to the maximum effective dose. The term "low-density cholesterol" refers to a metric, but the particle itself cannot be precisely measured.

References

Amarenc Rosengar A, et al. “Anterior inferior cerebellar artery territory infarcts. Mechanisms and clinical features.” Archives Neurology 1993;50:154 -161

Andy Jones, Graham Bentham “EPIC-Norfolk prospective population”; MRC epidemiology unit: 2012

Badimon L, Badimon JJ, Turitto VT, Vallabhajosula S, Fuster V. Mechanism of platelet aggregation using type one collagen to strengthen. A description of how trauma to vessel heals. Its impact on how blood cells move about, v WF& blood clotting and movement Circulatory physiology. 1988;78(6):1431–1442. [PMID: 3263902]

Badimon L, Badimon JJ. “Procedures for thrombus formation large arteries in nonparallel lines. Platelets aggregate and thrombus increases in size at the tip of the wall of arteries that are maximally affected.” J Clin Invest 1989; 84(4):1134–1144. [PMID: 2794050]

Boekholdt SM, Arsenault BJ “Mora S, the relationship between LDL cholesterol, non-HDL cholesterol, and apolipoprotein B levels a meta-analytical study indicating an increase in coronary events..” JAMA. 2012 Mar 28;307(12):1302-9. doi: 10.1001/jama.2012.366.

Bogousslavsky J, Regli F. “infarcts due to anterior cerebral artery in the registry by Lausanne”. Archives Neurology 1990;47:144â€“150.

Chapman M.J “is there relevance in the effect on low density lipoprotein cholesterol by statins?” European Heart Journal Supplements Volume 6: Issue supply: 710 2012.

Cholesterol in Adults “(Adult Treatment Panel III): Final Report.US Curr Atheroscler Rep. 2012” Apr; 14(2):130-4. doi: 10.1007/s11883-011-0224-x

Cholesterol in Adults “(Adult Treatment Panel III): Final Report.US Curr Atheroscler Rep. 2012” Apr;14(2):130-4. doi: 10.1007/s11883-011-0224-x

Elizabeth Barrett-Connor, MD “Sex Differences in Coronary Heart Disease” CIRCULATION;1997 95; 252-264.

“Expert Panel on Detection Evaluation, &Treatment of High Blood Fuster V. Cardiovascular disease and the United Nations Millennium Development Goals: a significant need for analysis.” Nat Clin Pract Cardiovasc Med 2006;3:401. [PMID: 16874332

Garg PR, Kaabita 1 S, “A study on cholesterol” Ann Human Biol: 2012 Nov 30.

Havarkate F, Thomson SG JR, Pepys MB. Synthesis of CRP &increased incidence of heart problems in angina with and without enzyme elevations: “European Concerted Action on Thrombosis and Disabilities Angina Pectoris Study” [NIH Publication No. 02- 5215. September 2002.] Circulation. 2002;106:3143–3420

Hosseini K, Saedeghian S, Mamoudian M, Hamiidian R, Abasi A. “Family history of cardiovascular disease as a risk factor for coronary artery disease in adult offspring.” Monaaldi Archieve Chest Disease. 2008 Jun;70(2):84-7.

Jeremy A. Kelley, MSN, RN, CRNP1 “CORONARY ARTERY DISEASE AND SMOKING CESSATION INTERVENTION” Online Journal of Health Care, vol. 9, no.2, Fall 2009

John C. M. Brust, Merritt's Neurology, “Cerebral Infarction”11th Edition, Lippincott Williams & [NIH Publication No. 02-5215. September 2002.] Circulation. 2002;106:3143–3420

KruthHS, “Sequestration of aggregated low-density lipoproteins because of macrophages”:CurrOpinLipidol: 2002; 13:483

Langile BL, “part in generation and progression of thrombus formation the circulatory system and atherothrombosis of heart”. In: Fuuster V, “Atherothrombosis and Coronary Artery Disease”, 2d edition Philadelphia: Lippincott Williams & Wilkins, 2005:561–568: Lancet 1997:349:462–466.

Liby P, “Act local, act global: Inflammation and the multiplicity of “vulnerable” coronary plaques.” J Am. ColCardiology 2005; 45:1600

Meenakshi Sharma, “Premature Coronary Artery Disease in Indians and its Associated Risk Factors” Vascular Health Risk Management. 2005 September; 1(3): 217–225.

Michal G Marrmota, “alcohol and heart disease” Int. J. Epidemiology. (2001) 30 (4): 724-729.

Michel Miller, MD, “What Are the Effects of Statins on Triglycerides and What Are the Results of Major Outcomes Studies?” JAMA. 2012 Mar 28;307(12):1302-9.

Mulvihill ER, Jager J, “Smooth muscle cells in atherothrombosis plaques have a distinct phenotype”. Arteriosclerosis Thrombosis Vascular Biology 2004; 24:1283

Napoli C, D'Armiento FP, Mancini FP, et al. “Mechanism of formation of the streak of fat in embryonic aorta & which causes severe increase because of increase in cholesterol levels in the mother increase in the concentration of LDL and how its metabolism by oxidation cause influx of monocyte at the site of very early atherogenic plaques”, J Clin Invest 1997;100:2680–2690. [PMID: 9389731]

Nilson J,” Atherogenesis regulated by immune mechanisms: prospects for the development of preventive medicines”. Arteriosclerosis Thromb Vascular Biol 2005;25:18–28. [PMID: 15514204.

Pasterrkamp G, “Expansive arterial remodeling: Location, location, location”. Arteriosclerosis Thromb Vascular Biol 2004; 24:650.Pp. C58-C63.

Rana JS, “The role of non-HDL cholesterol in risk stratification for coronary artery disease” Elsevier Ireland Ltd Nov 16:2012.

Sigdel M, “study on cholesterol” B.BMC Res Notes. 2012 Nov 17;5(1):640.

Stamler, J, “Do we have a grading between cholesterol levels and risk of dying prematurely? “Findings in 356,222 primary screenees of the “Multiple Risk Factor Intervention Trial (MRFIT)”: JAMA, 1986, 256:2823–2828. [PMID: 3773199]

Susan A, “Use of Statin" Lipid-Lowering Drugs Compared With Guidelines” JAMA January 2001:Vol 161, No.1234.

The Expert Panel. Third Report of the National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, & Treatment of High Blood Cholesterol in Adults (Adult Treatment Panel III). The end report.” Circulation, 2002, 106:3143–3421.

“Third report of the National Cholesterol Education Program (NCEP) : Tuzcu EM, “High prevalence of coronary atherosclerosis in asymptomatic teenagers and young adults: Evidence from intravascular ultrasound.”Circulation 2001; 103:2705.

Vaccaro JA KELLEY, New England journal of medicine 2012;2012:916816. doi 0.1155/2012/916816.

Robert C. Byrd, Sniderman AD,“Discordance analysis of Apolipoprotein B and non-high-density lipoprotein cholesterol as markers of cardiovascular risk in the INTERHEART study Atherosclerosis.” 2012 Dec;225(2):444-9.

To understand the reasons behind the higher occurrence of coronary heart disease in diabetes, a thorough investigation of how oxidized LDL from various population subgroups affects endothelium-dependent relaxation is necessary.